2017-06-302026-03-272017-06-302004María Teresa Cordero Ortíz0014-299910.1016/j.ejphar.2004.09.018https://doi.org/10.1016/j.ejphar.2004.09.018https://repositorio.inprf.gob.mx/handle/123456789/6793engacceso cerradoPeripheral involvement of the nitric oxide-cGMP pathway in the indomethacin-induced antinociception in ratarticle1879-07121-3Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz43-48Holanda503PIFIR modelIndomethacinAntinociceptionNitric oxideRatModelo PIFIRIndometacinaAntinocicepciónÓxido nítricoRataAnimalsAnti-Inflammatory Agents, Non-Steroidal-PharmacologyArginine-MetabolismChronic DiseaseCyclic GMP-PhysiologyDisease Models, AnimalEnzyme Inhibitors-PharmacologyFemaleGout-Chemically inducedGout-ComplicationsGout-PsychologyIndomethacin-PharmacologyNG-Nitroarginine MethylEster-PharmacologyNerve Tissue Proteins-Antagonists & inhibitorsNitric Oxide-PhysiologyNitric Oxide Donors-PharmacologyNitric Oxide Synthase-Antagonists & inhibitorsNitric Oxide Synthase Type INitroprusside-PharmacologyPain-Chemically inducedPain-Drug therapyPain Measurement-Drug effectsPeripheral NervousSystem-Drug effectsRatsRats, WistarSignal Transduction-PhysiologyUric Acid