Insulin-mediated upregulation of T-type Ca2+ currents in GH3 cells is mediated by increased endosomal recycling and incorporation of surface membrane Cav3.1 channels

2017-06-292026-03-272017-06-292012Mercedes Simoni Nieves0143-416010.1016/j.ceca.2012.06.005https://doi.org/10.1016/j.ceca.2012.06.005https://repositorio.inprf.gob.mx/handle/123456789/4618engacceso cerradoAnimalsBrefeldin A/pharmacologyCalcium Channels, T-Type/geneticsCalcium Channels, T-Type/metabolismCell Membrane Permeability/drug effectsEndosomes/drug effectsEndosomes/metabolismGene Expression Regulation/drug effectsGene Expression Regulation/geneticsHEK293 CellsHumansInsulin/metabolismMonomeric GTP-Binding Proteins/geneticsMutation/geneticsPatch-Clamp TechniquesPituitary Gland/cytologyPituitary Gland/metabolismRatsTranscriptional Activation/drug effectsTranscriptional Activation/geneticsrab GTP-Binding Proteins/geneticsInsulin-mediated upregulation of T-type Ca2+ currents in GH3 cells is mediated by increased endosomal recycling and incorporation of surface membrane Cav3.1 channelsartículo1532-19915Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz377-387Países Bajos52Ca2+ channelsInsulinCav3.1 channelsRab11Endosome recycling