2017-06-302026-03-272017-06-302010Juan Carlos Bautista Ramírez0013-722710.1210/en.2010-0279https://doi.org/10.1210/en.2010-0279https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2940536/https://repositorio.inprf.gob.mx/handle/123456789/6172engacceso cerradoContribution of TNF-alpha and nuclear factor-kappaB signaling to type 2 iodothyronine deiodinase activation in the mediobasal hypothalamus after lipopolysaccharide administrationarticle1945-71708Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz3827-3835Estados Unidos151AnimalsCyclic AMP Response Element-Binding ProteinmetabolismEnzyme Activationdrug effectsGene Expression Regulation, Enzymologicdrug effectsHypothalamusdrug effectsHypothalamusimmunologyHypothalamusmetabolismI-kappa B ProteinsgeneticsI-kappa B ProteinsmetabolismImmune Systemdrug effects Immune SystemmetabolismIodide PeroxidasegeneticsIodide PeroxidasemetabolismLipopolysaccharidesadministration & dosageLipopolysaccharidespharmacologyMaleNF-kappa BmetabolismNF-kappa BphysiologyPhosphorylationdrug effectsRatsRats, Sprague-DawleySignal Transductiondrug effectsSignal TransductionphysiologyThyrotropingeneticsThyrotropinpharmacologyTumor Necrosis Factor-alphametabolismTumor Necrosis Factor-alphaphysiologyCyclic AMP Response Element-Binding ProteinI-kappa B ProteinsLipopolysaccharidesNF-kappa BTumor Necrosis Factor-alphaThyrotropiniodothyronine deiodinase type IIIodide Peroxidase